BioMedsAI @BioMedsAI
AI Scientist in Biomedical and Clinical Research and Discovery! https://t.co/40q8wAVOHT [email protected] biomedsai.com Queens, New York City, USA Joined August 2026-
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@16vchq Happy to present BioMedsAI Inc. Always available -> [email protected] biomedsai.com
@thepaulthomson @OpenAI Hi... Here is our startup: biomedsai.com [email protected]
Our founder have published a phenomenal research on PPM1D in the @ASH_hematology @BloodPortfolio ! Recommended to read!
Read the paper: ashpublications.org/blood/article/… It’s my absolute pleasure and utmost honor to share our recent publication in the @BloodPortfolio of the @ASH_hematology , titled: “Molecular Consequences and Targeting of PPM1D Overexpression in Myeloproliferative Neoplasms,” with me as
@16vchq biomedsai.com We believe in serving humanity 🙏
@EmmaQian_ @nova_ai One of us can join if available!
@verbove biomedsai.com Is the one & only AI scientist in the market!
@OfficialLoganK We know, thats why we are contributing in Biomedical research and innovation! Can we do benchmarking of Argon ? Let us know... biomedsai.com [email protected]
@sarahzorah Can we get into @claudeai startup program ? biomedsai.com
It's our pleasure and honor to get accepted into @Google "@GoogleStartups " Program! Come , talk with us... biomedsai.com [email protected]
@MartinGTobias @aliansarinik Then, fund us in pre-seed , now... biomedsai.com
@eriktorenberg @cosign send me an invite... [email protected]
@1752vc Agentic AI in biomedical data analysis, new target discovery, drug design and disease specific virtual cell designing.
@DarioAmodei We are focused on BioMedical AI development and we agree with Dario. We need to be careful , evaluate at full extent and perform measured RSI. Thank Dario. Ready to work with you.
nature.com/articles/s4200… Excited to share our new publication at @CommsBio ( @Nature ), “Resistance to modulation of in vivo gene expression by genomic editing.” The study took long time and enormous effort of a whole team of investigators! We investigated whether CRISPR editing of KLF1 regulatory enhancers (EHS1 and INT1) could reproduce KLF1 haploinsufficiency and increase fetal hemoglobin (HbF/γ-globin)! 1. Although enhancer editing reduced KLF1 and increased γ-globin in primary human CD34+ cells, these effects were not consistently maintained following xenotransplantation—revealing an unexpected resilience of the KLF1 regulatory locus in vivo. 2. Our ATAC-seq studies suggest that dense regulatory architecture, enhancer redundancy, and long-range chromatin interactions can buffer gene expression even after efficient genome editing. This highlights an important principle for therapeutic genome engineering: editing an enhancer does not necessarily phenocopy natural genetic haploinsufficiency. Grateful to our collaborators and especially pleased to have contributed as a co–equal contributor to this work. These findings may help guide the design of more durable and predictable genome-editing strategies for hemoglobinopathies and other genetic diseases. #GenomeEditing #CRISPR #GeneTherapy #KLF1 #Hemoglobinopathies #FetalHemoglobin #Hematology #StemCells #Epigenetics #ATACseq #CommunicationsBiology
@biogerontology Building a virtual cell us far more complex in biology than 30 minutes with Astra. Come one, you know it.
DrDx @drdx007
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Asst. Prof. MD BABU M... @BabuuDx
518 Followers 82 Following Founder of BioMedsAI, Inc , Assistant Professor @IcahnMountSinai @MountSinaiNYC ; @IndianaUniv (alum) ; DM for Collaboration!
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muhammad ibrahim 🧬... @MD_IBRAHIM2211
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Blood Journals Portfo... @BloodPortfolio
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