Mohamed Kehili 🔬 @m_kehili
Master Student in Genetics 🧬 Ile-de-France, France Joined September 2016-
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Mohamed Kehili 🔬 retweeted
In most cells of your body, mitochondria are constantly in movement. They interact with one another like social creatures do, exchange information, sometimes fuse to become longer and bigger, and then fragment to die off and be recycled. Mitochondria move and are dynamic…
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Mohamed Kehili 🔬 retweeted
Type 2 diabetes is often considered a protein misfolding disease. But where are these toxic proteins found? 🤔 🚨In new work out today in @NatMetabolism, we show that mitochondrial protein misfolding (yes mitos🤯) leads to beta cell damage in T2D. 🚨 nature.com/articles/s4225… 1/n
Mohamed Kehili 🔬 retweeted
Mitochondrial origins of the pressure to sleep: nature.com/articles/s4158…
Mohamed Kehili 🔬 retweeted
Response of spatially defined microglia states with distinct chromatin accessibility in a mouse model of Alzheimer’s disease nature.com/articles/s4159…
Mohamed Kehili 🔬 retweeted
Taking the STING out of neurodegenerative disease rupress.org/jem/article/22…
Mohamed Kehili 🔬 retweeted
Connection and communication between the nervous and immune systems | Nature Reviews Immunology nature.com/articles/s4157…
Mohamed Kehili 🔬 retweeted
A Review published in Molecular Neurodegeneration discusses the many connections between UFMylation with Alzheimer’s disease pathogenesis, emphasizing the role of UFMylation in these pathways and their abnormalities in AD. …arneurodegeneration.biomedcentral.com/articles/10.11…
Mohamed Kehili 🔬 retweeted
Today, @NIH announced a new policy to cap how much publishers can charge NIH-supported scientists to make their work publicly accessible. This reflects our broader effort to restore public trust in public health by creating an open, honest, and transparent research atmosphere.…
Le monde des revues scientifiques au bord de l’asphyxie lemonde.fr/sciences/artic…
Mohamed Kehili 🔬 retweeted
Challenging a longevity dogma in muscle aging. Validated epigenetic clocks (pan-mammalian and muscle-specific) reveal a surprise: an 85% drop in NAD⁺ in mouse muscle leaves epigenetic age unchanged. Muscle structure, function, and mitochondria stay strong: NAD⁺ loss ≠ muscle…
Mohamed Kehili 🔬 retweeted
Nonuniversality of inflammaging across human populations | Nature Aging nature.com/articles/s4358…
Do scientists read enough fiction? | EMBO reports embopress.org/doi/full/10.15…
Mohamed Kehili 🔬 retweeted
Heat shock proteins function as signaling molecules to mediate neuron–glia communication via extracellular vesicles during aging in C. elegans @dong_yan nature.com/articles/s4159…
Mohamed Kehili 🔬 retweeted
Neuroinflammation across neurological diseases science.org/doi/10.1126/sc…
Mohamed Kehili 🔬 retweeted
Heat shock proteins function as signaling molecules to mediate neuron–glia communication in C. elegans during aging: nature.com/articles/s4159…
Mohamed Kehili 🔬 retweeted
Dysfunctional mitochondria trap proteins in the intermembrane space | The EMBO Journal embopress.org/doi/full/10.10…
Mohamed Kehili 🔬 retweeted
Seriously, understanding about the mitochondria and its basis in chronic disease is not a political thing. There’s good science that hard working scientists have published. theatlantic.com/health/archive…
Mohamed Kehili 🔬 retweeted
Exercise triggers protective responses in the dentate gyrus of an Alzheimer’s mouse model, boosting neurovascular astrocytes, microglia, and oligodendrocyte progenitors. Exercise may help mitigate AD-related cellular changes. nature.com/articles/s4159…
Mohamed Kehili 🔬 retweeted
Emerging role of mitochondrial calcium levels in cellular senescence and in switching cell fates | Nature Aging nature.com/articles/s4358…
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