Brilliant book. LIke Arthur Lydiard, Olympic 5000m runner, Dr Bakken MD, came to his unexpected conclusions from decades of intensive personal lactate testing to determine the training that produced the best long term effects. Training approach now followed by world class athletes including Norwegian Olympic gold medallists in different sports Jakob Ingebrigtsen (running), Kristian Blummenfelt (triathlon) and Johannes Klaebo (cross-country skiing).
Nearly 2,000 psychiatrists graduate annually without learning about metabolic approaches to mental health. Metabolic Revolution is closing this gap—one book at a time.
Support or share: gofundme.com/f/transforming…
Als de EU leiders naar Alden Biesen kwamen zonder echt plan voor reductie van energiekosten, herzien van klimaatdoelstellingen, ETS, antidumpingplan, afbouw van regeldruk dan waren ze beter thuis gebleven. #industrienood
EU censorship files. No debate allowed.
A Parliament that is not allowed to hold the EU commission accountable is not democracy - it's theatre
#EUcensorshipfiles@Jim_Jordan@elonmusk
Koen Schoors is Prof aan UGent en wil ons via de digitale euro dwingen om mee te gaan in zijn links groene fantasie dictatuur. Uiteraard krijgt hij een vrij podium bij zijn vriendjes van MSM. Zeer zorgwekkend.
New Zealand authorities arrested this man for exposing the government’s COVID vaccine data.
What he uncovered inside the vaccine batches was horrifying.
When Barry Young, a former Ministry of Health employee, examined the data, he was alarmed to find a 21% death rate tied to Batch ID 1.
Batch ID 1: Total Vaccinated 711, Death Count 152, 21.38% Dead
Batch ID 2 showed similar results, with a 17% death rate — Total Vaccinated 221, Death Count 38, 17.19% Dead
Batch ID 3 followed close behind, with a 15% death rate — Total Vaccinated 310, Death Count 48, 15.48% Dead
According to Young, New Zealand’s underlying mortality rate should be just 0.75%, meaning the odds of these outcomes occurring by chance are roughly 100 billion to 1.
And these weren’t isolated cases. Numerous other batches showed death rates of 4.5% and higher.
“So statistically, what we’re saying is that there is no chance that this vaccine is not a killer,” Young concluded.
And instead of triggering an urgent investigation, this data triggered something else entirely. 🧵
This is disturbing. I've purchased Kerrygold butter because I thought it was made from cows that only ate grass. It turns out that if a cow eats 1 blade of grass in its life the company can say the cow is grassfed. This was the basis of a lawsuit against Kerrygold.
The product needs to say 100% grass-fed to be sure the cow was always fed grass.
thecleanlivingpath.com/pollution/the-…
Member of Parliament of the United Kingdom confirms if they say anything negative about Islam they can now be prosecuted
Sarah Pochin “We could be prosecuted for saying anything critical of Islam — Are we going to find ourselves arrested and thrown into jail by this new elite police force that this government has set up to monitor our speech, our debate, our views. Well, I will see you in jail because I will never be silenced as I speak up for you.”
Some more obvious points that came to me overnight @drjamesdinic.
First I hope you have, by now, read and digested the essence of our review.
Second, you are absolutely correct about underpowered studies. This point was made more than 14 years ago by the Oxford Unit who, like you, argue that studies with perhaps less than 100 subjects are all underpowered.
bmjopen.bmj.com/content/2/4/e0…bmj.com/content/345/bm…bmj.com/content/345/bm…
However the reality is that for complex studies like ours journals.physiology.org/doi/full/10.11… it's impossible to recruit such a large number of subjects to completely alter their diet and rigorously stick to it for the period that is require by the study design. With respect, you don't do experiments so you wouldn't know this.
And where does one find the money to conduct such trials other than from industry who have a vested interest in finding only that which will increase their business? All our studies are funded internally to insure there is no external influence on what we find and what results we can publish.
So what should we do? Simply accept this and not do any research? Or try to design experiments which can still provide hard evidence despite this serious limitation?
As I will show this is exactly what we did. We overcame this problem through an innovative experimental design. We did this by comparing not just one intervention but two with the same subjects as I will explain shortly.
Third, a point you seem reluctant to grasp is this: Our trial was not a dietary intervention trying to determine whether or not a high- or a low-carbohydrate diet is superior (as you present it). It was a trial designed to determine whether muscle of liver glycogen content is more important for human exercise performance. So we used different dietary interventions to study this. But the dietary interventions were not the goal of the study. Instead we used two different dietary interventions in order to address the, until then, unanswered question: Is it liver or muscle glycogen content that is more important for human exercise performance? Of course the findings have important implications for the type of diet we might consider prescribing for different athletes. But that was not goal of the trial, nor of the review. So the animosity that the study has and will attract is misdirected. We're studying biology not human nutrition. Got it?
Fourth, the question of underpowered studies. The risk of underpowered studies is that they might miss a real effect because too few subjects are included in the trial. In other words they might miss an effect which is quite small. For example studies of the statin drugs require tens of thousands of subjects because the potential benefits that are being searched for are, clearly, quite small. But small "underpowered" studies can still detect a real effect if it is sufficiently large.
Our study that you critique at length, compared the effects of muscle and liver glycogen manipulation on human exercise performance using 10 subjects. We found no effect of muscle glycogen on prolonged exercise performance. But you are correct. Perhaps there is an effect that we missed. But if so, it is not an obvious (ie very large) effect.
Instead our "underpowered" study found an easily identifiable effect of liver (and blood glucose - the small glucose pool) glycogen on prolonged exercise performance which increased by ~20% with carbohydrate ingestion. I know of no other published dietary intervention that has been shown to produce an equivalent effect.
So yes - perhaps the study was underpowered to measure an effect of muscle glycogen on prolonged exercise performance and, if so, the effect is likely small (and perhaps related to increased muscle lactate production for liver gluconeogenesis, better to protect against hypoglycemia during prolonged exercise, following a period of eating a high-CHO diet).
But it was clearly not underpowered to detect the very much larger effect of small amounts of CHO ingested during exercise.
So which intervention is likely to be more important for human exercise performance? Clearly those that prevent premature hypoglycemia during prolonged exercise.
Exactly as our review which we started working on long before this experiment was planned, eventually confirmed would be the most likely outcome on the basis of the 16 lines of evidence we found in the published literature.
Hope that clears up a few remaining points of your, I am sure, well intended but wide-of-the-mark critiques.
@PhilipPrins11@AKoutnik@LoreofRunning1@BenBikmanPhD@drjasonfung@sweatscience@zoeharcombe@bigfatsurprise@lowcarbGP@PaulBLaursen@CarynZinn@theplews1@stevemagness@drjamesdinic@Scienceofsport@MetabolicUncle
Yes there are some studies showing this outcome. But all these studies are underpowered according to your guidelines. Also were the low carb group protected from developing hypoglycemia during exercise? And the majority of the studies performed in the last 5-10 years and which
Dr. Campbell and Professor Clancy Left Speechless
Peer reviewed science buried for 800+ days
Fake studies promoted
Peer reviewed science suppressed
No outrage
No coverage from legacy and mainstream media
Professor Robert Clancy criticizes an 800+ day delay of an "incredible" peer reviewed paper which would have dramatically changed the Pandemic trajectory. It's an OXFORD RCT showing chloroquine, hydroxychloroquine reduced PCR-confirmed symptomatic covid by at least 57%.
GROK AI add further weight to Clancy.... "a meta-analysis of 12 RCTs confirms moderate prophylaxis benefit, supporting claims of underutilized early prevention amid vaccine prioritization."
Results so good that Governments might have thought of mandating drugs when they forced messenger RNA
Remember that the Lancet published and later retracted this completely fake study that was used to discredit hydroxychloroquine and mislead people to think it was not effective and dangerous.
This was the most egregious medical and academic fraud in the history of the world.
This is absolutely mind blowing. The conspiracy nutters were right!
“Under the scheme, drivers would need a residents’ permit that allows 100 days of free travel per year through six traffic filters during operating hours.
Meanwhile, a separate permit allows 25 days of free travel per year through six congestion charge locations during charging hours, and after this, drivers face fines if they travel without the relevant permission.”
telegraph.co.uk/news/2026/01/2…
Eight days have passed since the Federal Court of Appeal upheld Judge Mosley’s 2024 decision that the invocation of the Emergencies Act, which led to the excessive use of force by law enforcement agencies from across Canada against peaceful protestors, was unlawful and unjustified.
NO statement from the PMO.
NO statement from the Justice Minister.
NO msm exclusive reports.
NO substantive statement from the official opposition.
NO apologies to Canadians for breaching their rights & freedoms in the last egregious of ways.
As culinary abominations, skimmed milk and low-fat dairy have been making meals miserable for far too long. Full-fat milk is back, and the world will be a better place 🥛🧀🐮🙌
The problem in science, as we show in the article, is when scientists "know" what is the truth and are unwilling to test the veracity of their knowledge. Up to 2010 I also "knew" that carbs were the key dietary macronutrient for human exercise performance. In 2017 @PhilipPrins11 and his team gave me the opportunity to test my "knowingness". Over 7 years of experiments we proved our "knowingness" was wrong. We discovered that many other labs around the world have reported exactly the same findings (as I point out in my upcoming debate with Professor Louise Burke due for publication in AJCN in due course. How can you simply ignore those findings as if they don't exist?) When we started we had no idea what the outcome would be. That I think is how science is meant to be tackled.
Also, to bring out the tired and unsubstantiated statement say that every athlete involved in high intensity exercise is eating a high carb diet, is nonsense. We know of teams in combat-type sports who have adopted the low carb diet with extraordinary results. For obvious reasons this is confidential information - they don't want to give away their advantage. I predict that in 5 years or so, every team in those sports will be eating the LCHF diet. Or they will no longer be competitive.
Your mind is closed. So it's pointless continuing this discussion. @AKoutnik@LoreofRunning1
@KetoCounselor@AKoutnik@ProfTimNoakes Nothing is broken.
We know high carb diets are better for high intensity exercise vs ketogenic diets.
If keto diets were actually better then every elite athlete would be on them but they aren’t.
There’s a reason why almost every elite athlete, especially sports athletes,
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Alumnus KU Leuven. Beschikbaar voor lezingen over klimaat realisme